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Subject: texinfo-pretest post from address@hidden requires approval
Date: Fri, 06 Jul 2007 12:22:36 -0500

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    Subject: Buck scientists saved the mice from their prescribed fate by a 
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--- Begin Message --- Subject: Buck scientists saved the mice from their prescribed fate by a blocking a newly discovered molecular pathway . Date: Sat, 7 Jul 2007 01:22:22 +0800 User-agent: Thunderbird 1.5.0.12 (Windows/20070509)
Brokers Move On ERMX!

EntreMetrix Inc. (ERMX)
$0.18

Heavy trading today as ERMX announced its launch of digital support
tools for its portfolio companies. Brokers are getting ahead of this
steady climb as they grab up large blocks of shares for there clients.
Look at the numbers and get on ERMX Friday morning!

It's the most noteworthy late stage of a process best halted earlier,
but at this point, too many people are suffering for lack of any port in
the storm - a breadth of approaches is vital.

Diabetics will undergo stem cell therapy to replace the islet cells in
their pancreas.

" Killing cells is easy.
The changes, recommended late last year by a government-commissioned
independent review, would permit therapeutic cloning under strict
controls. We can hope that this is the result of a rising tide of new
advances. Find some of the highlights - such as the latest from the team
working on those regenerating MRL mice - in the following Fight Aging!

If we can find drugs that can rescue these mitochondrial functions, that
would be a much better drug than the current therapies that are targeted
on dopamine replacement.

" Killing cells is easy. phpYou'll be seeing much more in the years
ahead as researchers are recruited and the faculty grows. This strikes
me as a direction that ought to get huge amounts of funding. And it can
stimulate the growth of new capillaries in the brain, heart and skeletal
muscles.
The hope is that stem cells from their own marrow, which have been grown
in culture, can repair the damaged area, avoiding the need for
replacement.

Scientists at Intercytex are trying to create a cell bank to allow cells
to be transferred from one individual to another.

ANTI-AGING, A TERM LOST TO THE JUNKYARD? The science of healthy life
extension. htmlHELP PROMOTE THE OPEN LETTER ON AGING RESEARCHThe
Scientists' Open Letter on Aging Research is not, in my opinion at
least, receiving the press attention it deserves.
Exactly what form that damage takes remains to be discovered, but we can
all hope that future biotechnology will render accidents of birth - and
aging itself - moot.

They showed drops in body temperature and blood-insulin levels - both
phenomena that have been seen in long-lived, calorie-restricted animals.

We can print any desired structure, in principle .

It's already a few years too late for that, especially given that some
varieties of therapy are already commercially available. Glenn
Laboratories for the Biological Mechanisms of Aging. But many people who
believe it will extend life practice calorie restriction to a lesser
degree.

'Bone marrow stem cells are now being used to repair hearts of people
who have had myocardial infarctions. Killing just the right cells is
very hard - but scientists are making good progress.

What we've found is that non-methylated genes that reside in a
particular suburb near methylated genes are also silenced.

Why set yourself up to miss out on the chance of a far longer, healthier
life? is aging, growing in population and getting fatter.
This gives us a better chance of clearly separating defects in
development from aging. htmlVia ScienceBlog, an argument for aging as an
evolutionary adaption rather than a tradeoff: "Aging is taken to be an
adaptation, but one that benefits the community, not the individual. But
it doesn't, and the scientific and advocacy communities have barely even
started on the long road to building such a thing. Early coverage and
reports from community blogs and local media are already online; you'll
find a selection linked in the following Fight Aging! Find some of the
highlights - such as the latest from the team working on those
regenerating MRL mice - in the following Fight Aging! phpCongratulations
are due to the Foundation volunteers and all the generous donors of the
past few years - working together, you have greatly advanced the cause
of healthy life extension research. Activism and advocacy.
nuclear-targeted hTERT, in the absence of mitochondrial localization, is
associated with diminished mtDNA damage, increased cell survival and
protection against cellular senescence.

But it doesn't, and the scientific and advocacy communities have barely
even started on the long road to building such a thing. Click here to
see how easy it is.

Elderly cells, it seems, cannot. But there's much more to look at as
well, such as telomeres in Bristlecone Pines and a closer look at the
dysfunctional aging immune system - and what could be done to fix it up.
eceFrom the Independent, a look at progress towards first generation
stem cell based therapies for hair regrowth: "Hair is grown by the
dermal papilla cells in skin. htmlRandall Parker makes an interesting
point here: "Imagine you could be told two or three or four decades in
advance what you are going to die from. is aging, growing in population
and getting fatter. All pointing the way to longer, healthier lives. "
This is fairly slow, standard old school drug development - and the
press release is for an early stage of that inch by inch process. " A
good job that researchers are already working on ways to replace
age-damaged mitochondrial DNA.

htmlRare acelerated aging syndromes have a great deal to teach us about
the biochemistry of aging and cancer. The Glenn Foundation For Medical
Research has deep pockets and its founders have a strong interest in the
long term - seeing this through and doing it right, step by step. " A
number of groups are working on the technologies required to manipulate
telomeres and telomerase - these tools will be most useful.

Why set yourself up to miss out on the chance of a far longer, healthier
life?

Government obstruction of freedom of medical research must cease if we
are to see significant advances in the future of healthy life extension
medicine. nuclear-targeted hTERT, in the absence of mitochondrial
localization, is associated with diminished mtDNA damage, increased cell
survival and protection against cellular senescence.

The researchers use "tailor-made tiny sponge-like nanoparticles laced
with the drug docetaxel.


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